Dilated Cardiomyopathy - A Medical Dictionary, Bibliography, by Icon Health Publications

By Icon Health Publications

In March 2001, the nationwide Institutes of future health issued the next caution: "The variety of sites providing health-related assets grows each day. Many websites supply precious info, whereas others can have info that's unreliable or misleading." moreover, as a result speedy raise in Internet-based info, many hours should be wasted looking, picking out, and printing.This publication was once created for doctors, scholars, and individuals of most of the people who are looking to behavior clinical examine utilizing the main complicated instruments to be had and spending the smallest amount of time doing so.

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Extra info for Dilated Cardiomyopathy - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References

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To elucidate signaling mechanisms, we will determine which of the major Gi pathways in the heart (Gi2 and Gi3) are involved; and whether signaling via the G protein alpha subunit and/or the betagamma dimer is involved. Using 3 model systems we will investigate Gi signaling effects (both deleterious and beneficial) and the ec-couplingand signaling mechanisms involved in: Aim 1. CHF caused by Ro1 expression; and recovery after terminating Ro1 expression. Aim 2. Acute Cardioprotection caused by Ro1 expression.

The central goals of this project are to identify genes involved in the pathogenesis of DCM, and to obtain the necessary training in cardiovascular genetics over a proposed five year period, to prepare for a career as an independent investigator. Research will focus on four specific alms critical for identification of genes involved in DCM pathogenesis. 1) Ascertainment and phenotypic characterization of individuals and families with DCM. Families and sporadic cases suitable for genetic analyses will be identified from congestive heart failure and transplant clinics at1he University of Utah and University of Pennsylvania system hospitals.

The overall hypothesis of this proposal is that mechanical factors play a significant role in the tissue remodeling associated with dilated cardiomyopathy and cardiac failure. Sophisticated computational models in conjunction with experimental studies in rodents with different etiologies of heart failure (both genetic and surgically-induced) will help elucidate the role of mechanical factors in the progression of cardiac dilation and failure. The following hypotheses will be tested: (1) Dilated cardiomyopathy and eventual heart failure are mediated by mechanical loads on the heart, and the transition from a compensated hypertrophic state to cardiac failure is dependent on a critical level of stress or strain.

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